Quick info
Ketamine is produced synthetically and used in medicine as an anaesthetic, among other things. Ketamine belongs to the group of dissociatives.
The effect of ketamine as an intoxicant is strongly dose-dependent: In lower doses, it has a disinhibiting and relaxing effect similar to alcohol, while higher doses can induce trance-like states up to out-of-body or near-death experiences (K-Hole). There may be a fragmentary dissolution of the environment and bodily sensation, thoughts may break off, feelings of weightlessness or floating may emerge. Sensory perceptions and space-time sensation change. At very high doses, detachment from one's own body and/or ego dissolution or fusion with the environment may occur.
After the trip: drowsiness, memory of the experience is often only partially possible.
The dosage of ketamine is highly dependent on the desired effect.
Snorted: 20 - 75 mg
Swallowed: 250 - 400 mg
Injected intramuscularly: 70 - 120 mg
Appearances
white crystalline powder. Rarely liquid as a solution.
Onset of action
Snuffed: after 5 - 10 minutes
Swallowed: after 15 - 20 minutes
Injected intramuscularly: within 2 - 5 minutes
Injected intravenously: within seconds
Duration of action
Depending on the dose and form of consumption 30 minutes - 3 hours
Movement and communication may be severely restricted. Partial or complete insensitivity to pain, coordination disorders, sensation of weakness, loss of appetite, nausea, vomiting, uncoordinated muscle movements, dizziness, slurred speech, increased pulse and blood pressure and cardiac arrhythmias. At high doses, muscle stiffness, paralysis and narcosis, at very high doses, epileptic seizures and coma.
Ketamine puts a strain on the cardiovascular system. A ketamine trip can be psychologically very stressful. Many ketamine users report near-death experiences, nightmare hallucinations, tunnel visions, blackouts and short periods of memory loss.
After repeated consumption within a short period of time, the effect diminishes considerably and a tolerance develops.
Long-term risks
Ketaminecan cause dependence with psychological symptoms. Chronic use damages the liver and kidney and can lead to depressive moods and anxiety. It is thought that even in small doses, ketamine can cause dysfunction in areas of the brain responsible for memory, learning, and cognition. The more often it is consumed and the larger the individual doses, the more alarming these disturbances become.
Ketamine is not a party drug! Do not take ketamine alone and make sure you are in a familiar and comfortable environment. Allow enough time to process the trip in peace afterwards.
Dose carefully. Small differences in dose can cause significant differences in effect.
Take regular breaks from consumption.
You need a place to sit or lie down, because at high doses there is a great risk of collapsing or fainting.
Because of the insensitivity to pain, you can hurt yourself without noticing it.
Do not take ketamine on a full stomach.
People with cardiovascular problems, high blood pressure or glaucoma should absolutely refrain from consuming ketamine!
Refrain from mixed consumption. When combined with alcohol, benzodiazepines or opiates, there is a risk of respiratory arrest! Mixing with uppers (e.g. cocaine) can lead to circulatory problems, increase in heart rate and shortness of breath.
Increased risk behaviour due to narcotic effect and loss of reality.
LSD analogues are substances that are chemically very similar to LSD and can have comparable effects. Some of them have been known for a long time (e.g. ALD52, ETH-LAD, AL-LAD, PRO-LAD etc.) and have been studied pharmacologically as well as psychopharmacologically, at least in part. Others are newer "creations" (e.g. the derivatives 1P-LSD,1B-LSD, 1cP-LSD, 1V-LSDetc.), for which only few or no data are available. Certain LSD analogues can (still) be legally produced, traded and consumed in some countries, which is the main reason for their distribution.
Most LSD analogues are naturally different from LSD in their effect and/or potency (e.g. ETH-LAD, AL-LAD, LSZ etc.). In contrast, the so-called 1-acylated LSD compounds (e.g. 1P-LSD, 1V-LSD, 1B-LSD, ALD-52, etc.) are presumed, on the basis of pharmacological studies, to convert into LSD in the body (they function as so-called prodrugs) and thus have a comparable psychoactive effect to LSD.
In the case of prodrugs of LSD and LSD analogues, it has not been conclusively clarified whether, in addition to their psychoactive effect, they can produce other pharmacological effects. How potent these prodrugs are compared to the resulting substance (e.g., 1P-LSD to LSD), and to what extent a delay in onset of action occurs in each case, may be substance-dependent and cannot be generalized. Therefore, it is important to approach the dose/effect carefully to avoid overdoses.
If you or someone else needs urgent help after taking drugs or alcohol, call an ambulance on 144. Tell the emergency responders everything you know.
It could save lives.